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Gabapentin 300 precio for at least 4 weeks; and 1% lidocaine 30 days in two sessions. After the completion of drug treatment, patient was invited to participate in the fMRI study. pre- and post-treatment groups, patients were scanned twice by a blinded psychologist (Z.L.X.) who was to the treatment group. In this study, patients underwent an fMRI scan after a three day period of drug treatment; the patient group participated in fMRI scan twice. The primary finding of our study is that a chronic treatment with psilocybin can reduce the functional connectivity between right prefrontal and temporal regions (F3/F4; MNI coordinates, [−22, −6]). The authors note that results do not indicate which of the two brain areas is responsible for the decreased connectivity. The authors also note that it is important to note that the results from pre- and post-treatment scans were highly correlated. They state that this "correlation may be due to the fact that two groups were already highly integrated, and the effects of pre post-treatment may be attributed to a long-lasting and highly integrated effect of the drug treatment." Additionally, they comment on their current findings, stating that "the observed reduction in the connectivity between right prefrontal and temporal areas may not be related to the reduction in hallucinations" (p 622). "Thus, we cannot exclude the possibility that observed reduction in functional connectivity could be related to the subjective effects of drugs or to changes in cognition. Conclusion There has been increasing evidence relating psilocybin therapy to alterations in subjective experiences of psychotic patients. As mentioned above, the purpose of this study was to identify brain structures that are associated with the subjective effects of psilocybin treatment and to investigate the effect of chronic treatment on connectivity across these brain regions. Overall, the authors observed